Prediction of hemodynamics blood flow in healthy and stenotic aortic valve by the fluid- structure interaction method

Aortic stenosis is caused by narrowing of the orifice of the aortic valve. Stenosis is described by calcium deposition on the leaflets. With the growth of stenosis, hemodynamics, mechanical performances and blood flow through the valve are changed. This study proposes the comparison of two new fluid-structure interaction of healthy and stenotic aortic valve finite element code ADINA during a complete cardiac cycle. Due to the hardening of calcified aortic valve, the orifice area decreased from 2.4 cm2 for the healthy AV to 1.4 cm2 for the stenosis case. The axial velocity and mean pressure gradient increased in mid systole. In addition, strain concentration and higher stress values were observed on the leaflets in stenotic valve than healthy case. Pressure distribution and velocity results were in good agreement with echocardiography data in published literatures. Although improvements are still needed, our computed data were well simulated closing and opening of the healthy and the calcified aortic valves.